Innate immune signaling
Endosomal TLRs, MyD88, NLRP3 and TLR9-mediated immune activation.
- Mechanistic evaluation
- Disease-model research
- Translational milestones
We welcome discussions with pharmaceutical companies, biotechnology partners and academic groups interested in immune signaling modulation and regenerative peptide therapeutics.
A collaboration can begin with a program, a mechanism or a complementary experimental capability.
Endosomal TLRs, MyD88, NLRP3 and TLR9-mediated immune activation.
FAP1, VMP3, SDP-4 and SP1 programs in tissue repair research.
Address a defined biological question or experimental capability.
Examine complementary expertise and evidence needed for later decisions.
Consider a specific program and the rights relevant to it.
Identify the program or research area, the evidence of interest and the scientific contribution each organization could make.